Unlock peak brain power: How 20 mins of HIIT triples BDNF!
📚 References
─────────────────────────────────
[1] Therapeutic Effects of miR-29b-Enriched Nanovesicles on Mitochondrial Injury and Neuroinflammatory Processes in Alzheimer’s Disease. | Gönüllü S, Dalkılınç E, Çelik O et al. (2026)
→ Alzheimer’s disease (AD) is defined by the accumulation of extracellular amyloid-β plaques and intracellular neurofibrillary tangles, which drive neuronal and synaptic impairment. In AD, the accumulation of protein aggregates leads to neuro
[2] Catalpol protects against MPP(+)-induced neurotoxicity by targeting PINK1/DJ-1-mediated mitophagy and the TrkB/Akt/BDNF/Bcl-2 axis. | Yang HB, Chen YH, Nhung NTT et al. (2026)
→ Parkinson’s disease (PD) is a prevalent neurodegenerative disorder characterized by dopaminergic neuronal death of unclear etiology. While levodopa remains the gold standard for managing PD motor symptoms, it lacks disease-modifying efficac
[3] Oral exposure to non-brain-penetrable microplastics induces neurotoxicity via disrupting the gut microbiota-tryptophan metabolism-microglial autophagy cascade. | Wu Y, Dong Z, Wang D et al. (2026)
→ The neurotoxic potential of microplastics (MPs) is an emerging environmental health crisis. However, the majority of environmental MPs are unable to penetrate the blood-brain barrier (BBB), leaving their mechanism of neurotoxicity largely u
[4] Polysaccharide-driven gut microbial metabolism in neurodegenerative brain diseases: from SCFAs and tryptophan catabolites to neuroinflammatory regulation. | Peng C, Chen L, Wu Z et al. (2026)
→ The gastrointestinal tract constitutes the principal anatomical interface of the microbiota-gut-brain axis (MGBA), orchestrating neuroimmune and metabolic crosstalk. Dysregulation within this network drives neurodegenerative pathogenesis th
─────────────────────────────────
🔬 AI Health Lab
Breaking down the latest science on aging, longevity & health.
#Shorts #aging #longevity #AI #health #biohacking #science
source